Barium chloride produces excitatory effects on neuronal signaling through mobilization of intracellular Ca2+. This has been correlated to causing increases in potential difference and short-circuit currents in stripped sheets of rat intestine with subsequent effects on intestinal secretion. The toxic effects of Ba2+ are mediated through neuronal excitation, and sub-toxic administration of Barium Chloride produces arrhythmias in animals. Barium Chloride-promoted arrhythmias have been employed in studies investigating antiarrhythmic compounds. Barium Chloride demonstrates selective inhibition of MMP-2 (matrix metalloproteinase 2), suggested to proceed through displacement by Ba2+ of a Ca2+ ion at the catalytic center of MMP-2.
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氢氧化铜，CP，95%
3,4-二甲氧基苯甲酸 9...
S-碘化丁酰硫代胆碱 98%
S-联萘酚 99%
α,α,α-三氟苯乙酮 99...
苯基三乙氧基硅烷 ,98%
苯甲醇 ,AR,≥99%
正辛酸 CP,98%
辛酸钠 99%
氯化镁，AR，98%
2-氨基-3-(三氟甲基)吡...
α-松油醇 ，GC≥96%
苯丁酸，AR,97%
4-硝基吲唑，97%
4-氯咪唑，97%
聚(4-苯乙烯磺酸钠)，av...
2-溴苯甲胺，98%
N-羟基正辛酰胺，98%
2,2 -双噻吩，98%
4,4 -二溴二苯甲酮，97...
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